Blockade of histone deacetylase inhibitor-induced RelA/p65 acetylation and NF-κB activation potentiates apoptosis in leukemia cells through a process mediated by oxidative damage, XIAP downregulation, and c-Jun N-terminal kinase 1 activation

Yun Dai, Mohamed Rahmani, Paul Dent, Steven Grant

College of Medicine and Health Sciences

Virginia Commonwealth University
Virginia Commonwealth University Health System
VA Commonwealth University

Research output: Contribution to journal › Article › peer-review

223 Scopus citations

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Blockade of histone deacetylase inhibitor-induced RelA/p65 acetylation and NF-κB activation potentiates apoptosis in leukemia cells through a process mediated by oxidative damage, XIAP downregulation, and c-Jun N-terminal kinase 1 activation

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Biochemistry, Genetics and Molecular Biology

Pharmacology, Toxicology and Pharmaceutical Science

Immunology and Microbiology